Most of the blood glucose that a diabetic makes is coming directly from their liver, not their food. So, you can only "eat to your meter" in one of two ways:
- Get a clear panel for your stomach, so you can actually see how much glucose your liver is about to dump. *This might ruin your appetite causing you to eat less, overall, and inadvertently become calorie restriction.
- Avoid putting more glucose into a system that is already overrun with glucose.
I recommend #2. It's cheaper, easier and less gross. But even so, doing this, still doesn't mean that the body will magically acquire proper blood glucose regulation. Instead of "eating to your meter", use your meter to see how your body regulates its blood glucose instead.
2. Can eating chicken cause you to become "hypoglycemic"?
I know there are very "special snowflakes", out there, that claim to have a physiology, which baffles scientists around the world. But, the reality is, that nothing mysterious is happening to them. If no doctor has been able to tell them what's going on, which I doubt is the case, I will do it myself and I'm not even a doctor.
People who are overweight/obese, have metabolic dysfunction. Forget all the BS they are trying to sell you about how "healthy" they are, despite the "extra weight on their body". Forget about all the lab work they present, where they only show you the results they want you to see. Forget about all the "keywords" they want to throw at you like "lower inflammation" and "lower cholesterol". All of it is irrelevant. Forget all about the medications they are simply refusing to take, rather than actually not requiring them. All you have to remember is that overweight/obese people have metabolic dysfunction because they have blood glucose dysregulation, which deteriorates insulin expression/action over time. They have excess body fat which causes a very high insulin demand on the body, furthering blood glucose dysregulation.
When the body is under a high insulin demand, it always maintains a chronically high, basal insulin level. This causes for insulin to stop functioning properly. Insulin is supposed to function in pulses. It is not suppose to be chronically high, while never rising high enough to stop catabolism or dropping low enough to avoid resistance. The body reacts to this abnormality in insulin function by keeping its blood glucose "set point" to high. The body actively tries to do this by ramping up its counter regulatory stress systems to release blood glucose in response to any lowering of blood glucose and to any fluctuation in insulin release.
Metabolic dysfunction causes for the body to do anything and everything to keep its blood glucose high. It will break down its lean body mass to do this. It will release stored glycogen, from the liver, to do this. It will convert everything that's eaten into glucose, to do this. It will do whatever it takes to continue making glucose and keep its serum glucose levels high in order to match basal insulin. Your body is dosing glucose as a response to abnormal insulin expression.
When someone with metabolic dysfunction consumes protein, which is a macronutrient that raises insulin, but not blood glucose, one of two things usually occurs:
- The glucose floodgates open, but there is not enough insulin to close them. Now the body releases its own blood glucose, at an exaggerated rate, causing high postprandial blood glucose, known as hyperglucagonemia. This is usually the case for people who have prediabetes or diabetes. By this point, they have lost their first phase insulin response, due to beta cell damage and/or loss. There is simply not enough of an insulin spike to regulate the release of glucagon, after eating, and the body catabolizes itself with no control.
- The body fails to release enough glucose in order to counteract insulin release and now too much glucose is being cleared too quickly, from the bloodstream, and there is a significant dip in blood glucose. This can almost be described as hypoglucagonemia, This may be the result of insulin resistance of the alpha cells. Even though no clinical hypoglycemia occurs, the body has symptoms as if there was. This is usually the case for people who have metabolic syndrome, but have not yet crossed over into prediabetes or diabetes. These people have not yet lost their first phase insulin response, but you can already see a problem with their glucagon to insulin ratio. This has been well documented in insulin assays, the most famous ones were by Dr. Joseph R. Kraft.
The way to resolve this is to stabilize blood glucose regulation, improve insulin sensitivity and leptin expression, which will cause a loss of body fat. It's not to double down on "keto" or whatever else hasn't worked, isn't working and won't magically begin working. The solution is not to avoid protein. It's not to avoid chicken. It's not to eat more dietary fat. It's not to blame "stress". It's not to blame the diet industry or "trauma". It's blood glucose dysregulation that's driving this problem, not anything else.
That's why you cannot be healthy while carrying "extra weight on your body". "Extra weight on your body" is equal to "metabolic dysfunction". It's the way the body tries to prevent diabetes. So, when you have excess body fat, you are always at diabetes's door. The person must lose body fat in order to reduce the insulin demand, on the body, which is further disrupting proper blood glucose control.
3. Can high blood pressure be controlled without prescriptions?
High blood pressure is a very serious condition that can lead to cardiovascular disease (CVD) and/or damage. It has to be monitored and managed by a healthcare professional.
Some people with metabolic syndrome experience a "pseudo high blood pressure". This is high blood pressure of no underlying pathology, except massive water retention caused by high insulin levels. Insulin retains and holds water in the body, causing a rise in blood pressure, which disappears once the person lowers their insulin. This is why "losing weight" reduces blood pressure.
Other causes for high blood pressure will not be addressed by merely lowering insulin levels and so blood pressure will continue to remain high. For this reason, if your blood pressure remains high, even after adopting a low carb diet and lowering your insulin levels, you must talk to your healthcare professional and get on a treatment plan to keep it under control.
4. "Keto" will you lose body fat and gain muscle.
If the ketogenic protocol you are following is designed to burn body fat, then you will lose body fat. If it's designed to just lower blood glucose, by addressing your jellybean habit, then no. You won't lose an inch.
Ketogenic protocols are not designed nor intended for the gain of muscle mass, though many people combine these diets with weight training. But, technically, only adequate protein and exercise achieves muscle gain.
5. Can the increase in blood lipids due to following "keto" result in "cholesterol in the eye"?
There are two benign conditions, which are often referred to as "cholesterol in the eye":
- Arcus Senilis - a bluish ring along the outer edge of the cornea.
- Xanthelasma - minor fatty deposits on the surface of the skin, usually along the eyelids.
The third condition, Hollenhorst Plaque, is not benign. This is a buildup of plaque that has broken off from a clot “upstream,” usually from a much larger artery, such as the carotid artery. This plaque will then block the flow of blood of another artery, further “downstream", causing death to the tissue that is fed by it. This is called a Branch Retinal Artery Occlusion (BRAO) or, if the main arterial supply to the retina is blocked, a Central Retinal Artery Occlusion (CRAO).
All the cells in our body need oxygen to survive, and your arteries are the highways that carry that oxygen. If these “highways” are blocked, blood doesn’t reach the tissues it was intended for, and neither does the oxygen. This results in irreversible loss of function, which could include blindness, of the affected eye, depending on the location of the blockage. A retinal artery occlusion is essentially a “stroke” of the eye. Worse still, the presence of a Hollenhorst Plaque, increases the likelihood that another plaque could also find its way to the brain.
All of this isn't caused by "high cholesterol", though you may have high total serum cholesterol numbers. But, this also occurs to people with low or normal cholesterol. This is actually caused by a genetic lipid function abnormality that puts you at high risk for cardiovascular disease and/or stroke, regardless of total cholesterol numbers. There are also other complex factors involved in this pathology, as malfunctioning lipids are not the sole factor.
That's the important thing to keep in mind because you don't want to be lulled to sleep with "lower cholesterol" and think you are no longer at risk or are "cured". This genetic condition is not resolved by simply "lowering cholesterol", though that's the only tool conventional medicine offers at this time. It is better addressed by changing your lipid composition, through diet and certain medications, alongside conventional treatments in order to reduce your actual risk.
So, make sure you see an expert, like a cardiologist and/or lipidologist, to receive professional help in treating this condition and not rely solely on your primary doctor. If you don't, you can find yourself having serious health problems in the future.
6. "Inflammation" will prevent weight loss.
No. Inflammation causes "water weight" retention, not fat retention.
The person who is having an inflammatory response has a negative interplay between cortisol and insulin, but this does not prevent fat loss. This can make fat loss difficult, because of the effects it has on blood glucose control, but it doesn’t prevent fat loss. Difficulty is not equal to inability. The only thing that truly "prevents fat loss" is leptin dysfunction.
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