Welcome


My name is Gina and I would like to welcome you to my blog!

On this blog, I not only share the dietary and lifestyle approach which reversed my metabolic disease and achieved my weight loss, but I also debunk many misconceptions surrounding obesity and its treatment.

I am 5'5" and was weighing 300 lbs., at my heaviest. I lost a total of 180 lbs. I went through several phases of low carbohydrate dieting, until I found what worked best and that is what I share on this blog. Once on a carbohydrate restricted diet, along with intermittent fasting, I dropped all of the weight in a little over two years time.

My weight loss was achieved without any kind of surgery, bariatric or cosmetic. I also did not take any weight loss medications or supplements. I did not use any weight loss program. This weight loss was solely the result of a very low carbohydrate, whole foods based diet, along with daily intermittent fasting and exercise.

I allow discussions in the comments section of each post, but be advised that any inappropriate or off-topic comment will not be approved.

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Mar 9, 2020

Six common beliefs addressed, Part 63

1. Will eating lots of fat stop hunger and cause weight loss?

First, weight loss does not occur from lack of hunger. At least, not the type of weight loss you are trying to achieve which is specifically weight loss from fat loss. That only occurs through the stabilization of blood glucose. 

"Hunger for fat" is a sign of metabolic disease. I want that to be completely understood and clear. Having to eat enormous quantities of fat, in order to reach satiety, is a sign that you are very metabolically sick and not better or getting better by any stretch of the imagination. People with healthy metabolisms do not ever experience this. They reach satiation with protein, not the energy macronutrients.

Metabolically dysfunctional people experience this same issue with carbohydrates. They have to eat an enormous amount of carbs in order to reach satiety, significantly disrupting their blood glucose and making them uber fat. When they switch to fat, the same thing occurs. This is a sign that leptin is not communicating fat stores, to the brain, and the body is constantly in starvation mode, since it doesn't see its own exceeded fat reserves.

This is active obesity. The body is demanding large amounts of energy, through hunger, which it obviously doesn't need, in order to continue building fat mass. Hunger for fat or hunger for carbs, means you are getting fatter. If the diet was truly reversing your obesity and healing your metabolism, this wouldn't be occurring. Stop feeding your fat mass with fat. You already did that before with carbs.  

2. Calories is the same as energy, so there is no need to differentiate between the two.

I know that this can be confusing, but it's a good way to begin thinking differently about food. The term "energy" helps take into account the different macronutrients and how they are partitioned and prioritized, by the body, since the body doesn’t have a linear “caloric” relationship with food.

Calories are a piss poor way to measure energy in an effective way, as far as metabolism goes, regardless of how practical it may be. This is because, ultimately, the body is primarily affected by the allocation of energy, not its measurement. The body partitions calories depending on the macronutrient they come from. This means that knowing the total calories, of a food item, tells you nothing about whether it is obesogenic or not. You can be on a very low calorie diet that’s still fattening. As an example, roughly speaking, 100 calories of dessert go primarily to your fat (m)ass, through the disruption of blood glucose, while 100 calories of steak goes primarily to your lean muscle mass because there is no blood glucose disruption.

3. The only thing that matters is 'absolute risk' not 'relative risk.'

"Risk" has to do with statistics and it refers to probabilities, not certainties. It’s a very complex subject and you shouldn’t get bogged down by it, as it will not help you lose a pound.

Pharmaceutical studies always use relative risk, rather than absolute risk, since they only want to compare between those that took the drug, being studied, and those that didn’t. This means that if statins reduce your relative heart disease risk by 50%, that’s only relative to those that didn’t take the pill. But, if your absolute risk for heart disease was only 2%, to begin with, then the statin only decreased your individual risk to 1%. That’s still relatively 50%, but not very impressive overall. This is why it’s often said that absolute risk is really all you should care about. The problem is who determines it?

Individual absolute risk is extremely difficult to determine, so even though it may be the only thing that truly matters, it’s unusable in practice. Though absolute risk can be calculated, in a group of people participating in a study, the results will only pertain to them, not you. Absolute risk may vary depending on the pool of people being used. Maybe another group of people, even though similar, might have a different absolute risk result. After all, there are people who smoke till old age, without any lung disease. Others eat birthday cake daily, without any diabetes. Others have all their family members die of heart attacks, but they never develop the disease themselves.

To eliminate all confounders, relative risk is commonly used, as it narrows things down to only two groups of people - those who took the drug and those who didn’t, eliminating all of the other individual unknowns. The media may distort the results, to get readers attention, but the actual studies have all the data disclosed and it’s not difficult to obtain them.

Having said all of that, since our purpose here is to help with obesity and metabolic disease, and not create statistical reports on research studies, all of this is irrelevant. Focus on tried and true biology, along with your results, and leave all these word play shenanigans alone.

4. Glucose is an electrolyte.

Glucose is not an electrolyte. It is usually added to electrolyte drinks, because it helps withhold electrolytes in the body better, due to its affect on insulin, but it is not needed.

5. You should separate sugar and glutamine, in meals, to prevent cancer.

Your own body makes glucose (sugar) and glutamine. Both of these are not essential nutrients, so they are not derived solely from diet. The body makes both abundantly. Glutamine is the most abundant amino acid in the body. Both glucose and glutamine are ancient nutrients that we evolved to use in multiple ways, so we make them ourselves. You can not separate them, in the human body, and separating them, in a meal, is ignorant and useless.

Instead of quackery, please learn real science.

6. You can not enjoy life or entertain yourself, without an occasional treat.

Some people are so dysfunctional, that they can't seem to live their lives happily without involving the mouth. I suggest you find an activity that is not dependent on the mouth. If you are bored and can't find entertainment, with life, do not take it out on your fat mass. Find something to do that doesn't require chewing and swallowing. There is just so much to do besides eating, that I would need a book to list it all.

Stop associating food with "party time". You don't need "treats". You are not a pet. Humans are way more complex than pets. That's why humans do not like to be kept by other humans, as pets, even with the promise of unlimited treats. The human brain usually does not find fulfillment in a life of treats, unless something is very wrong. You need food, not treats.

Eating a meal should be pleasant enough as is. It's just one of the many pleasurable things you can do in a day. Any day you aren't starving is a good day. You should never use food as a recreational drug.

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